Dermatitis: Difference between revisions

Line 179:

| [[File:Histopathology of lichen amyloidosis with Congo red.jpg|thumb|190px|Congo red.]]

|}

Interface dermatitis with lichenoid inflammation, not otherwise specified, can be caused by lichen planus-like keratosis, lichenoid actinic keratosis, lichenoid lupus erythematosus, lichenoid GVHD (chronic GVHD), pigmented purpuric dermatosis~~, lichen amyloidosus~~, pityriasis rosea, and pityriasis lichenoides chronica.<ref name="Alsaad2005"/> Unusual conditions that can be associated with a lichenoid inflammatory cell infiltrate are HIV dermatitis, syphilis, mycosis fungoides, urticaria pigmentosa, and post-inflammatory hyperpigmentation.<ref name="Alsaad2005"/> In cases of post-inflammatory hyperpigmentation, it is important to exclude potentially harmful mimics such as a regressed melanocytic lesion or lichenoid pigmented actinic keratosis.<ref name="Alsaad2005"/>

Interface dermatitis with lichenoid inflammation, not otherwise specified, can be caused by lichen planus-like keratosis, lichenoid actinic keratosis, lichenoid lupus erythematosus, lichenoid GVHD (chronic GVHD), pigmented purpuric dermatosis, pityriasis rosea, and pityriasis lichenoides chronica.<ref name="Alsaad2005"/> Unusual conditions that can be associated with a lichenoid inflammatory cell infiltrate are HIV dermatitis, syphilis, mycosis fungoides, urticaria pigmentosa, and post-inflammatory hyperpigmentation.<ref name="Alsaad2005"/> In cases of post-inflammatory hyperpigmentation, it is important to exclude potentially harmful mimics such as a regressed melanocytic lesion or lichenoid pigmented actinic keratosis.<ref name="Alsaad2005"/>

====Psoriaform dermatitis====

Examining multiple deeper levels is recommended if initial cuts do not correlate well with the clinical history.<ref name="Alsaad2005"/>

{|class="wikitable"

! Main conditions<ref name="Alsaad2005"/> !! Characteristics !! Micrograph !! Photograph

|-

| ~~Interface dermatitis~~ || ||

! Generally/Not otherwise specified

| Typical findings:<ref name="Alsaad2005"/>

*Regular epidermal hyperplasia, elongation of the rete ridges, hyperkeratosis, and parakeratosis.

*Usually:A superficial perivascular inflammatory infiltrate

*Often: Thinning of epidermal cells overlying the tips of dermal papillae (suprapapillary plates), and dilated, tortuous blood vessels within these papillae

| ||

|-

! Psoriasis

| *Clubbed rete pegs

* Acanthosis<ref name="Kunz2009">{{cite journal|vauthors=Kunz M, Ibrahim SM |title=Cytokines and cytokine profiles in human autoimmune diseases and animal models of autoimmunity|journal=Mediators of Inflammation|year=2009|volume=2009|pages=1–20|pmid=19884985|pmc=2768824|doi=10.1155/2009/979258}}</ref> <ref name="Raychaudhuri2014">{{cite journal |vauthors=Raychaudhuri SK, Maverakis E, Raychaudhuri SP |title=Diagnosis and classification of psoriasis|journal=Autoimmunity Reviews|volume=13|issue=4–5|pages=490–5|date=January 2014 |pmid=24434359|doi=10.1016/j.autrev.2014.01.008}}</ref>

*The stratum granulosum layer is often missing or significantly decreased

*Parakeratosis<ref name="Raychaudhuri2014"/>

| [[File:Micrograph of psoriasis vulgaris.jpg|190px]] || [[File:Psoriasis on elbow.jpg|190px]]

|-

| ~~Psoriaform~~ dermatitis ~~|| ||~~

|}

A psoriasiform dermatitis, not otherwise specified, may be caused by seborrheic dermatitis, psoriasiform drug eruption, chronic fungal infections, lichen simplex chronicus, chronic spongiotic dermatitis, secondary syphilis, pellagra and other nutritional deficiencies, pityriasis rubra pilaris, pityriasis lichenoides chronica, acrodermatitis enteropathica, inflammatory linear verrucous epidermal naevus, and rarely subacute psoriasiform cutaneous lupus erythematosus.<ref name="Alsaad2005"/>

|}

@@ Line 179: / Line 179: @@
 | [[File:Histopathology of lichen amyloidosis with Congo red.jpg|thumb|190px|Congo red.]]
 |}
-Interface dermatitis with lichenoid inflammation, not otherwise specified, can be caused by lichen planus-like keratosis, lichenoid actinic keratosis,  lichenoid lupus erythematosus, lichenoid GVHD (chronic GVHD), pigmented purpuric dermatosis, lichen amyloidosus, pityriasis rosea, and pityriasis lichenoides chronica.<ref name="Alsaad2005"/> Unusual conditions that can be associated with a lichenoid inflammatory cell infiltrate are HIV dermatitis, syphilis, mycosis fungoides, urticaria pigmentosa, and post-inflammatory hyperpigmentation.<ref name="Alsaad2005"/> In cases of post-inflammatory hyperpigmentation, it is important to exclude potentially harmful mimics such as a regressed melanocytic lesion or lichenoid pigmented actinic keratosis.<ref name="Alsaad2005"/>
+Interface dermatitis with lichenoid inflammation, not otherwise specified, can be caused by lichen planus-like keratosis, lichenoid actinic keratosis,  lichenoid lupus erythematosus, lichenoid GVHD (chronic GVHD), pigmented purpuric dermatosis, pityriasis rosea, and pityriasis lichenoides chronica.<ref name="Alsaad2005"/> Unusual conditions that can be associated with a lichenoid inflammatory cell infiltrate are HIV dermatitis, syphilis, mycosis fungoides, urticaria pigmentosa, and post-inflammatory hyperpigmentation.<ref name="Alsaad2005"/> In cases of post-inflammatory hyperpigmentation, it is important to exclude potentially harmful mimics such as a regressed melanocytic lesion or lichenoid pigmented actinic keratosis.<ref name="Alsaad2005"/>
 ====Psoriaform dermatitis====
+Examining multiple deeper levels is recommended if initial cuts do not correlate well with the clinical history.<ref name="Alsaad2005"/>
 {|class="wikitable"
 ! Main conditions<ref name="Alsaad2005"/> !! Characteristics !! Micrograph !! Photograph
 |-
-| Interface dermatitis ||  ||
+! Generally/Not otherwise specified
+| Typical findings:<ref name="Alsaad2005"/>
+*Regular epidermal hyperplasia, elongation of the rete ridges, hyperkeratosis, and parakeratosis.
+*Usually:A superficial perivascular inflammatory infiltrate
+*Often: Thinning of epidermal cells overlying the tips of dermal papillae (suprapapillary plates), and dilated, tortuous blood vessels within these papillae
+|  ||
+|-
+! Psoriasis
+| *Clubbed rete pegs
+* Acanthosis<ref name="Kunz2009">{{cite journal|vauthors=Kunz M, Ibrahim SM |title=Cytokines and cytokine profiles in human autoimmune diseases and animal models of autoimmunity|journal=Mediators of Inflammation|year=2009|volume=2009|pages=1–20|pmid=19884985|pmc=2768824|doi=10.1155/2009/979258}}</ref> <ref name="Raychaudhuri2014">{{cite journal |vauthors=Raychaudhuri SK, Maverakis E, Raychaudhuri SP |title=Diagnosis and classification of psoriasis|journal=Autoimmunity Reviews|volume=13|issue=4–5|pages=490–5|date=January 2014 |pmid=24434359|doi=10.1016/j.autrev.2014.01.008}}</ref>
+*The stratum granulosum layer is often missing or significantly decreased
+*Parakeratosis<ref name="Raychaudhuri2014"/>
+| [[File:Micrograph of psoriasis vulgaris.jpg|190px]] || [[File:Psoriasis on elbow.jpg|190px]]
 |-
-| Psoriaform dermatitis ||  ||
+|}
+A psoriasiform dermatitis, not otherwise specified, may be caused by seborrheic dermatitis, psoriasiform drug eruption, chronic fungal infections, lichen simplex chronicus, chronic spongiotic dermatitis, secondary syphilis, pellagra and other nutritional deficiencies, pityriasis rubra pilaris, pityriasis lichenoides chronica, acrodermatitis enteropathica, inflammatory linear verrucous epidermal naevus, and rarely subacute psoriasiform cutaneous lupus erythematosus.<ref name="Alsaad2005"/>
 |}

Dermatitis: Difference between revisions

Navigation menu

Search